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challenge lies in distinguishing genetic from epigenetic effects and establishing a causal relationship between DNA methylation variants and gene expression. In a collaborative project between Utrecht
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will contribute to the development and evaluation of gene therapy approaches for PMLD1. Your work includes: establishing and using isogenic iPSC-derived myelinating organoids as a human in vitro disease
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and progresses? In this PhD project, you will use innovative data analysis approaches and unique, deeply phenotyped cohorts to investigate the complex relationship between dietary factors, metabolic
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relationship between DNA methylation variants and gene expression. In a collaborative project between the Netherlands Institute of Ecology (NIOO-KNAW) and Utrecht University (UU), we will tackle these challenges
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affecting the white matter of the central nervous system. With the introduction of newborn screening and the first gene therapies, affected children can increasingly be identified and treated before symptom
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: identifying factors associated with parental distress, coping and resilience, drawing on developmental and psychological research. Long-term outcomes: investigating how these factors relate to quality of life
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preference, we encourage you to apply. This is what you will do You will investigate interactions among maternal factors, the infant gut microbiota and intestinal health to uncover underlying mechanisms. Using
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epigenetic editing delivered via lipid nanoparticles; Using a combination of cell models and human donor livers, you will investigate how these technologies can improve organ quality and increase the number
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manufacturing approaches, investigating melt behaviour, densification, defect formation, and microstructure in materials. A distinctive element of the project is its data-driven approach. Together with colleagues
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Are you passionate about human movement and developing clinical tools for understanding human movement disorders? Job description Challenge: Over a quarter of a million people in the