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Field
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Khmelinskii in collaboration with Jan Padeken offers the following PhD project: The progressive deterioration of protein homeostasis (proteostasis) is a primary hallmark of aging. As organisms age, the
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, and Jeffrey H. Chuang. 2012. “Transcriptional Enhancers in Protein-Coding Exons of Vertebrate Developmental Genes.” PloS One 7 (5): e35202. https://doi.org/10.1371/journal.pone.0035202 . Although cis
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to develop a protein purification facility. The IBGC operates under the supervision of the CNRS (INSB) and the University of Bordeaux. Its activities are dedicated to fundamental research in biology. The unit
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helps build the brain? Ribosomes are complex molecular structures that play active roles in protein synthesis. During development, progenitor cells generate a diverse pool of neurons that form the adult
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extracellular contractile protein translocation nanomachines. Importantly, the unique mode of action of T6SS allows bacteria to deliver folded proteins across membranes of neighboring bacterial or eukaryotic
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related to staff position within a Research Infrastructure? No Offer Description Assess the involvement of Factor H-related proteins (FHRs) accumulation on ECM stability and endothelial cell homeostasis
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the complex composition of dairy matrices (water, fat, proteins, minerals), give rise to heat-induced chemical reactions (Maillard reaction, caramelization and protein denaturation/aggregation). These reactions
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project in a collaborative research environment and to gain experience in a broad range of state-of-the-art experimental approaches. INFORMATION ABOUT THE RESEARCH PROJECT α2δ proteins are abundantly
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of additional datasets (RNA-seq, ChIP-seq, ATAC-seq, protein interactome, etc.) to investigate the impact of lamin mutations on spatial genome organization in lipodystrophy patient-derived cells. You will be
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The International Institute of Molecular Mechanisms and Machines Polish Academy of Sciences | Poland | 25 days ago
Postdoctoral fellow in the group of Prof. Peter G. Schultz at Scripps Research in La Jolla on the reprogramming of Botulinum proteases for targeted degradation of intrinsically disordered proteins