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Helmholtz Zentrum München - Deutsches Forschungszentrum für Gesundheit und Umwelt | Stein bei N rnberg, Bayern | Germany | 21 days ago
. The Institute for Diabetes and Cancer (IDC) at Helmholtz Munich aims to elucidate the molecular mechanisms linking metabolic dysfunction, diabetes, and tumorigenesis. The research group Metabolic Programming
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Thesis based on ABIS. The main areas have so far been studies on the development of Type 1 diabetes, but also studies on celiac disease, JIA, IBD, autism, obesity, allergy and recently also psoriasis
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, diabetes and cancer; see https://data.snf.ch/grants/grant/189065 ii) developing and validation of novel chemical probes and degraders to dissect mechanisms of adverse effects in cancer and metabolism. For a
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Primary Duties & Responsibilities: Information on being a postdoc at WashU in St. Louis can be found at https://postdoc.wustl.edu/prospective-postdocs-2/ . Trains under the supervision of a faculty mentor
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development resources. Details are available at: https://postdocs.yale.edu/postdoc-benefits Application Submission: Application materials should be submitted by email to [email protected] with
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at Lund University Diabetes Centre, one of the strongest centers for T2D research in the world. The position is within the research group Neuroendocrine Cell Biology at CRC in Malmö. The group consist of 2
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resources, including salary and outstanding benefits, that support your training and success at Iowa. https://grad.uiowa.edu/postdoctoral-affairs/postdoc-resources Education Requirement: PhD degree in a
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of phosphoinositide 3-kinase (PI3K) signaling in allergy, metabolic control, obesity, diabetes and cancer; see https://p3.snf.ch/project-189065 ii) developing and validation of novel chemical probes and degraders
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Researcher (R2) Positions Postdoc Positions Application Deadline 31 Jul 2026 - 12:00 (Europe/Prague) Country Czech Republic Type of Contract Temporary Job Status Full-time Hours Per Week 40 Offer Starting Date
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program seeks to increase our understanding of the immunological and molecular mechanisms involved in autoimmune dysregulation in Type 1 diabetes (T1D). Through the use of novel murine models of T1D, human