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Field
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project focuses on characterising leukodystrophy mouse models and evaluating promising proof-of-concept interventions. You will conduct longitudinal studies in mouse models, with a particular focus on
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assess their effects on myelination and oligodendrocyte survival. The most promising oligodendrocyte-targeting AAV variants will subsequently be tested in a PMLD1 mouse model to determine their therapeutic
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van Til at the Amsterdam Leukodystrophy Center (ALC). This doctoral project focuses on the preclinical assessment of gene therapy approaches in leukodystrophy mouse models for vanishing white matter
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and cellular functions. This project will combine complementary approaches in cellular neurobiology, molecular biochemistry, and developmental biology, using primary cultures of mouse neurons and cell
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biology approaches to identify transcriptional networks and signalling pathways controlling cardiovascular development. Using novel datasets generated from mouse models of congenital heart defects, the
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biology approaches to identify transcriptional networks and signalling pathways controlling cardiovascular development. Using novel datasets generated from mouse models of congenital heart defects, the
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samples from patients with multiple sclerosis and experimental mouse models. Conduct molecular and cellular biology experiments, including nucleic acid extraction, qPCR, chromatin profiling and
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research program investigating the molecular and developmental mechanisms underlying testicular dysfunction and infertility in a Wnt4 conditional knockout (Wnt4-cKO) mouse model. This position offers
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, and internalization studies. Plan, perform, and analyze preclinical biodistribution, PET, and SPECT imaging studies in mouse models. Investigate pharmacokinetic and pharmacodynamic properties of novel
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with mouse and human cell models, cell culture We require: A university degree in biology, biochemistry, or a related field Hands-on experience with the above techniques and methods Ability to work