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pathways are affected. We have recently shown that the mitochondrial phenotype in fibroblasts derived from both PWS and CMS22 patients can be rescued by overexpressing PREPL, providing the first evidence for
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? The University of Colorado offers a comprehensive benefits package. To see what benefits are available for Post-Doctoral Fellows, please visit: https://www.cu.edu/docs/benefits-package-guide-post-doctoral-fellows
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in bioenergetic assessment and mitochondrial function documented by scientific publications will be valued. VII.IV - All candidates who meet the admission requirements are subject to the academic
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. Preferred Qualifications Previous research experience in metabolism, cancer biology or mitochondrial biology. Experience with basic molecular biology and cell culture techniques (cell line propagation, cell
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FOR A POSTDOCTORAL RESEARCH FELLOWSHIP (BIPD) UNDER THE PROJECT “MITOCONEXÃO: INVESTIGATING THE MITOCHONDRIAL LINK IN ONCOCARDIOLOGY” (REF. NORTE2030-FEDER-02713500), CURRENTLY UNDERWAY AT THE ABEL
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biliary atresia, alpha-1 antitrypsin deficiency, Alagille syndrome, mitochondrial hepatopathies, cystic fibrosis, NAFLD, parenteral nutrition-associated cholestasis, acute liver failure, chronic cholestatic
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is contingent upon continued funding availability. The successful position will lead an research program focused on elucidating the mechanisms that drive mitochondrial dysfunction in neurodegeneration
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including barrier function, calcium signaling, and mitochondrial metabolic dysfunction studied in a novel 3D flow model and validated mouse models of hemorrhagic shock; and (3) multiomic (proteomic
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degradation in the nucleus. Proc. Natl. Acad. Sci. 116, 25828–25838 (2019). Decoding temporal windows for mitochondrial intervention across species using data-driven approaches Supervisor: Alberto Sanz Project
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functions and how these metabolic adaptations may be targeted therapeutically. Our research employs cell signaling and metabolism, fibroblast-immune cell crosstalk, metabolomics, mitochondrial biology