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(e.g. vividness, emotional intensity and perceived uncontrollability) are most clinically relevant and through which cognitive mechanisms they contribute to emotional distress and the persistence of mood
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methylation maintenance machinery, inducing genome-wide hypomethylation to identify genes regulated by methylation. Deploy an advanced, inducible Suntag-TET1cd epigenomic editing system to target specific
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. This project will be performed within the Neurotoxicology Research Group, which investigates the cellular and molecular mechanisms of action of potentially toxic substances on the nervous system. You will work
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treatment, identifies maladaptive patterns, and examines key underlying mechanisms. Your job As a PhD candidate you are responsible for the clinical part of this project. You will design studies, coordinate
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experiments that will reveal the physical mechanisms of transport and burial of microplastics and POC in turbidity currents down submarine canyons; incorporate these mechanisms into an existing computer model
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they will: perform scaled-down laboratory experiments that will reveal the physical mechanisms of transport and burial of microplastics and POC in currents down submarine canyons; incorporate these mechanisms
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the PI (Joris Eggenhuisen). Together they will: perform scaled-down laboratory experiments that will reveal the physical mechanisms of transport and burial of microplastics and POC in turbidity currents
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candidates and the PI (Joris Eggenhuisen). Together they will: perform scaled-down laboratory experiments that will reveal the physical mechanisms of transport and burial of microplastics and POC in currents
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to target DNA methylation maintenance machinery, inducing genome-wide hypomethylation to identify genes regulated by methylation. Deploy an advanced, inducible Suntag-TET1cd epigenomic editing system