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-induced senescence undergo permanent cell-cycle arrest and release a secretome ( https://doi.org/10.1016/j.celrep.2023.113181 ), which causes paracrine senescence and attracts immune cells to eliminate
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): analysis of cellular phenotypes, proliferation, viability, and cell death. * Omics data analysis: experience in the processing and analysis of omics datasets using R would be an advantage, particularly
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Erasmus MC (University Medical Center Rotterdam) | Rotterdam, Provincie Zuid-Holland | Netherlands | about 2 months ago
-blocking DNA damage can therefore cause cellular dysfunction and cell death, eventually resulting in DNA damage-induced aging and genome instability. Cells counteract these deleterious effects by
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cellular and molecular mechanisms that underlie the first steps of cell dissemination from the primary tumour. This will involve developing methods to live image tumour cell dissemination from primary
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Fundación Investigación Biomédica del Hospital de la Princesa | Madrid, Madrid | Spain | about 2 months ago
cell death, PPR.CAIXA.2023 HR23-00837. This contract will be funded by the funds from the aforementioned call associated with project PPR.CAIXA.2023HR23-00837. Eligibility criteria Scoring Criteria
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by spectral flow cytometry and scRNAseq, while CD4+ T cell / B cell interactions will be probed by confocal microscopy. The project will then aim at devising a new generation LO chip model that better
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the hypothesis that engineering AAV vectors to target specific cell-surface receptors can dramatically enhance gene delivery, enabling dose reduction, and improved safety. The planned research builds
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the hypothesis that engineering AAV vectors to target specific cell-surface receptors can dramatically enhance gene delivery, enabling dose reduction, and improved safety. The planned research builds
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%); assays for the evaluation of reactive species production (15%), glutathione determination (15%), and cell death assays, particularly by flow cytometry (10%). Motivation letter (20%). In the case
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. We focus on the innate immune response to Mycobacterium tuberculosis (Mtb) infection as it is the leading cause of death from a single pathogen worldwide. Our goal is to understand the host response to