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assays of nerve regeneration and pain signaling, and multi-omics technologies. By bridging fundamental neuroscience, stem cell biology, bioengineering, and clinical translation, our laboratory seeks
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Massachusetts Institute of Technology (MIT) | Cambridge, Massachusetts | United States | 2 months ago
experimental stem cell biology, human and mouse model systems, and machine learning approaches for phenotyping, target discovery, and therapeutic evaluation. Will be hired by the MIT Jameel Clinic for Machine
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pluripotent stem cells 3. Microscopy – immunofluorescence, confocal, wide-field, live cell 4. Microfluidics – bioengineering, device preparation, input/output bioanalysis 5. Genome editing – guide RNA
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to cell sources including adult stem/progenitor cells and immune cells for treatment of specific cancers, tissue degeneration, and autoimmune diseases. This interdisciplinary team comprises engineers
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experimental stem cell biology, human and mouse model systems, and machine learning approaches for phenotyping, target discovery, and therapeutic evaluation. Will be hired by the MIT Jameel Clinic for Machine
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, Stem Cell Biology, Biotechnology, Biochemistry, or a closely related discipline. Candidates with experience in bioinformatics and omics data analysis are encouraged to apply. Required Related Experience
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of Experimental Medical Science within the Faculty of Medicine is currently conducting research on the mechanisms underpinning the epigenetic control of the repetitive genome in stem cell models of early human
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interdisciplinary, multi-centre project aiming to identify antigens targeted by T cells mediating Graft-versus-Host Disease (GvHD) and Graft-versus-Leukaemia (GvL) following stem cell transplantation (SCT) as a
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Long-term culture-initiating cell (LTC-IC) assays or xenotransplantation-based stem cell assays. - Sequencing & genomics support including Library preparation for next-generation sequencing (targeted
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Biol 2010, Nat Protoc 2011, Stem Cell Reports 2014; Gascon et al, Cell Stem Cell, 2016). Notably, we have recently shown in the context of drug-resistant epilepsy that reactive glial cells can be