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with technology transfer structures, to promote dissemination to the clinical and industrial sectors. Open Science: The project fully adheres to an open science approach. Results will be preferentially
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17 Jul 2026 Job Information Organisation/Company INSERM Department Departement des Ressources Humaines Research Field Biological sciences Researcher Profile Established Researcher (R3) Leading
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16 Jul 2026 Job Information Organisation/Company INSERM Department neurospin Research Field Neurosciences » Other Researcher Profile Other Profession Positions Research Support Positions Application
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17 Jul 2026 Job Information Organisation/Company INSERM Department Departement des Ressources Humaines Research Field Biological sciences Researcher Profile Established Researcher (R3) Leading
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17 Jul 2026 Job Information Organisation/Company INSERM Department Departement des Ressources Humaines Research Field Biological sciences Researcher Profile Established Researcher (R3) Leading
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osteogenic phenotype. Attention will be paid to the design of microenvironments combining structural supports, biochemical signals (growth factors, morphogens), and potentially biophysical stimuli (mechanical
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the structure-function relationship of protein fragments in relation to extracellular matrix replacement. This job is based at the Laboratory of Tissue Biology and Therapeutic Engineering (LBTI – UMR CNRS 5305
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French National Institute for Health Research (INSERM) | Paris 15, le de France | France | 2 months ago
Understanding how genes and molecular signals coordinate tissue morphogenesis during organism development is crucial for progress in regenerative medicine and tissue engineering. The implications
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, jointly supported by Inserm, CNRS and the University of Strasbourg. The laboratory develops integrated research programs combining molecular biology, functional genomics, genome engineering, single-cell and
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Cardioembolic Stroke Risk Stratification using AI Accelerated Patient-Specific Blood Flow Simulation
and calcium release. However, links between mechanical stresses and LA structure, function, and hemodynamics remain unclear, as does the impact of myocardial fibrosis on LA hemodynamics and