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Field
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the goal of advancing pre-clinical findings toward clinical application. The fellow will conduct experiments involving transgenic and knockout mouse models to study osteoblast and osteoclast differentiation
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range of models including cell line, small animal, and respiratory organoid models — to enable pathogen agnostic activation of innate immunity. Key Responsibilities : Utilize cell line and transgenic
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-- Maintain and manage satellite animal rooms during and outside of experiments -- Manage, maintain, and breed multiple transgenic animal lines for experimental use -- Administrative support including
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, transport, and resistance, and potentially assess candidate functions using CRISPR and transgenic approaches. Anticipated Division of Time Research (55%): The Postdoctoral Associate will play a leading role
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analysis, and sex-differentiation pathway studies. Experience with multi-omic data integration and/or chromatin biology. Experience generating or working with transgenic insect lines, CRISPR/Cas9 editing
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. • Experience generating or working with transgenic insect lines, CRISPR/Cas9 editing, or RNAi-based functional validation. • Prior success in obtaining independent fellowship funding, or a clear plan and
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for Researchers: Transparent, open, and merit-based recruitment. (Available at: https://www.cicancer.org/about-cic/hrs4r The recruitment of research/technical staff by the Foundation is in accordance with
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University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | about 1 month ago
scientist to study the role of laminin-binding integrins in lung development, alveolar homeostasis, and alveolar repair. We utilize lung-specific transgenic mouse models along with ex vivo models, in vitro
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working environment for all laboratory staff • Maintain adequate stock of lab supplies and ensure proper setup and functioning of equipment •Manage a transgenic mouse colony, including but not limited
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combination of transgenic mice, -omic technologies, and organoid models to develop and refine hypothetical mechanisms through which epithelial progenitor biology is disrupted. The position will explore