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project 1. Only 2% of the human genome sequence codes for proteins, while most of it consists of noncoding sequences, including regulatory factor binding regions, transposable elements, pseudogenes
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identity and extracellular signaling, but how cell types, signaling mechanisms, and transcription factors jointly determine tissue structure is incompletely understood. The Koplev lab is recruiting
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-level computational model of the human cell. The program is led by Jan Ellenberg, Director of SciLifeLab and Mathias Ulhen, Director of the Human Protein Atlas, together with a collaborative, multi
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leading the investigation of structural and biophysical properties of miniaturized tumor environment models. A multidisciplinary approach is expected, integrating microfabrication, cell component and
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existing genomic datasets and enable analysis of gene regulation at cell-type resolution. The project places particular emphasis on ensuring high data quality and developing robust methods that can be
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for cardiometabolic and liver diseases. Current therapeutic strategies largely operate at the gene level, overlooking the functional diversity generated by alternative splicing. This project addresses this critical gap