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contribute to hereditary cancers and pediatric malignancies (https://bcchr.ca/maxwell-lab). Key areas include: • Mechanisms of cell division fidelity, spindle orientation, and cell polarity • Role of
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molecular and tissue level, and how its disruption alters epithelial – immune interactions that contribute to hereditary cancers and pediatric malignancies (https://bcchr.ca/maxwell-lab). Key areas include
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epithelial – immune interactions that contribute to hereditary cancers and pediatric malignancies (https://bcchr.ca/maxwell-lab). Key areas include: • Mechanisms of cell division fidelity, spindle orientation
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. The Research Environment The position is hosted jointly by the CoSAXS beamline and the Edler goup at Lund University. CoSAXS ( https://www.maxiv.lu.se/beamlines-accelerators/beamlines/cosaxs/ ) is a
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/organisation/geohyd and https://www.mn.uio.no/geo/english/research/groups/remotesensing ). We are a growing, lively, friendly and collaborative research environment in cold regions sciences, in
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through active learning. The scientific scope spans strain engineering, adaptive laboratory evolution, solid- and liquid-state fermentation, sensory and consumer research, and pre-clinical and clinical
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, Department of Geosciences Posting Number req26900 Department Geosciences Department Website Link https://geo.arizona.edu/ Location Tucson Campus Address Tucson, AZ USA Position Highlights The Atterholt
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University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | about 14 hours ago
evolution across tumor progression; (2) defining epigenetic mechanisms that drive diverse cancer and immune cell behaviors and therapeutic responses, using our innovative approaches such as Paired-TF
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UCSB, including the REAL AI initiative, which focuses on developing AI-powered digital twins for complex systems, the Departments of Physics, Ecology, Evolution and Marine Biology, Psychology and
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the Rehm Lab at GBI. The Rehm Lab, led by Principal Investigator Fabian Rehm, is building next-generation platforms for continuous evolution. We engineer orthogonal replication systems that drive