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. This NIH-funded position focuses on defining how germline gain-of-function mutations activate chronic inflammatory signaling, impair hematopoietic stem cell function, and drive progression to myeloid
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driven treatments for chronic pain and alcohol use disorder – two major unmet clinical needs. Our lab integrates behavioral pharmacology, molecular pharmacology, cell based assays, and mass spectrometry
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microenvironment. The project will employ genetically engineered mouse and human glioma models, along with advanced imaging techniques, single-cell and spatial transcriptomics, and molecular biology approaches
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