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will support and conduct simulation modeling research related to tobacco and lung cancer prevention and control. Duties include: Conducts data analysis using secondary data sources, such as national
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Sahoo and perform molecular/cellular studies of neuronal development using in vivo (mouse models) and in vitro (cultured primary neurons) models, involving skilled surgical procedures on mice and rats
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will support and conduct simulation modeling research related to tobacco and lung cancer prevention and control. Duties include: Conducts data analysis using secondary data sources, such as national
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options Employee and dependent educational benefits Life insurance coverage Employee discounts programs For detailed information on benefits and eligibility, please visit: http://uhr.rutgers.edu/benefits
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. The postdoctoral associate will receive mentored training in environmental epidemiology, cancer survivorship, exposure assessment, survival analysis, mixture modeling, biomarker analysis, and health disparities
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analysis, and/or numerical modeling. Experience in modeling and evaluating the impacts of climate and climate change, large-scale data analysis and interpretation, and independent research is required. City
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of transcriptomic, metabolomic, proteomic changes in rat/mouse models of Dementia. The individual will be responsible for planning and executing experiments independently. The individual has to be highly skilled in
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that include a mix of research design, fieldwork, data science, simulation modeling, writing, and presentations. The overarching goal of the Postdoctoral Associate will be to systematically investigate, evaluate
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independent research aligned with the aims of the Addiction & Decision Neuroscience Lab (ADN). Current work focuses on cognitive modeling of decision-making in both laboratory tasks and real-world settings, as
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structural biology (cryoEM), proximity labeling (TurboID), phosphoproteomics, and mouse models (including bGHTg acromegaly mice and breast cancer xenografts), we aim to uncover fundamental mechanisms of IGF1R