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osteoarthritis (PTOA)-associated joint degeneration and pain. A PhD is required Prior experience working with murine models of PTOA-associated cartilage catabolism and pain using pharmacologic treatments Onsite
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for genomics (e.g., generative models, transformers, agentic workflows) and/or statistical learning (e.g., network & spatiotemporal modeling, functional/longitudinal data, time-series). Analyze single-cell
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characterizing the endocrine-disrupting potential of plastic additives, with a primary emphasis on androgen receptor (AR) and estrogen receptor alpha (ERα) modulation, using established in vitro and in vivo models
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-throughput genomic screening approaches, primary cell culture systems, and animal models of influenza disease. For more information visit https://mgm.duke.edu/faculty-and-research/primary-faculty/nicholas
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advanced epidemiologic and biostatistical methods, including causal inference, survival and longitudinal modeling, and approaches for integrating multi-level and high-dimensional data. There will be
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, motivations and discoveries: https://sites.duke.edu/silverlab/ . We are especially interested in candidates excited to contribute to a new NIH program project with other Duke labs on kinase signaling across
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brain evolution. We employ a multifaceted strategy to bridge developmental neurobiology, RNA biology, and evolution. Learn more about our interests, motivations and discoveries: https://sites.duke.edu