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Sciences, University of Copenhagen, investigates how mitochondrial dysfunction, cardiolipin metabolism, and endocrine stress signals drive skeletal muscle decline during aging. We combine mouse models of
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Mechanisms of Aging and Metabolic Health Employer Personalwerk GmbH Location 10117, Mitte (DE) Salary E13 Closing date 11 Oct 2026 View more categories View less categories Discipline Health Science Job Type
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a highly motivated Postdoctoral Fellow to investigate the molecular genetics and neurobiology underlying microglial and oligodendrocyte function in the context of aging, neurodegeneration, and
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contribute to disease development following viral infections and during aging. We do so by bridging human cohort studies with mechanistic molecular biology by integrating longitudinal clinical datasets
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control, aging, and neurodegenerative diseases Maintaining protein homeostasis is essential for cellular function, and its progressive disruption is closely associated with aging and neurodegenerative
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The Cologne Graduate School of Ageing Research (CGA) in Germany is a joint venture of the University of Cologne Excellence Cluster on Stress Responses in Aging-Associated Diseases
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context of regeneration, aging and metabolism of bone and craniofacial tissues. Strong expertise and research track record of cellular and molecular biology, mechanobiology. Design and implement
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contribute to aging processes and resilience. We welcome applications from recent PhD graduates who are interested in these or related fields, particularly those who may bring a new technology or
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University of California, Los Angeles | Los Angeles, California | United States | about 20 hours ago
lab This research opportunity is centered upon interrogating the role of acquired genomic damage in tissue aging and age-related pathology. The Vandiver lab uses emerging genomic technology, induced
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Albert Einstein College of Medicine (Bronx, NY) | New York City, New York | United States | 19 days ago
, translational, and clinical studies to uncover mechanisms linking inflammation, DNA damage, and stem cell aging to cancer initiation and progression. Using murine and humanized models, human patient