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Max Planck Institute for Biology of Ageing, Cologne | Cologne, Minnesota | United States | 2 months ago
motivation for ageing research Candidates with very good English skills (C1 level, written and spoken) Application period: August 11 - November 3, 2026 Online application: https://application.ageing
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University of Cologne / Max Planck Society | Köln, Nordrhein-Westfalen | Germany | about 23 hours ago
The Cologne Graduate School of Ageing Research (CGA) in Germany is a joint venture of the University of Cologne Excellence Cluster on Stress Responses in Aging-Associated Diseases
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Your Job You will develop and evaluate model-based charging and vehicle-to-grid strategies for electric vehicles. Your work will combine battery and battery ageing models with real-world mobility
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Doctoral Researcher Position in Regeneration and Ageing Biology, Group Pentinmikko Employer UNIVERSITY OF HELSINKI Location Helsinki, Finland Salary The starting salary will be approximately 2600
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collaborative project on workplace well-being in elder care (WELLCARE) funded by the Working Environment Research Fund (AMFF). Description of the PhD project Within the context of ageing societies and
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Pentinmikko laboratory invites applications for a 3-year DOCTORAL RESEARCHER position in regeneration and ageing biology under the supervision of Dr. Nalle Pentinmikko. The successful applicant will
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the field of ageing. The Aboriginal and Torres Strait Islander Grant supports evidence-based knowledge about ageing in Indigenous Australian communities, to inform policy and program development. As
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development of reproducible wide-bandgap perovskite absorbers and complete perovskite subcells, including their intrinsic stability, phase behaviour, interfaces, and accelerated ageing. CEA focus
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<span lang="en">German Center for Neurodegenerative Diseases (DZNE)</span> | Bonn, Nordrhein-Westfalen | Germany | 4 days ago
ageing, preclinical changes and disease, as well as the comprehensive assessment of risk factors driving the onset and progression of both preclinical and clinical disease. These include genetic, molecular
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discovered that the toxin induces DNA damage responses in cultured cells that activates a senescence tumour suppressor mechanism ( https://doi.org/10.1038/s41467-019-12064-1 ). Cells undergoing toxin