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model, we were first to show that maternal obesity alone causes offspring cardiac structural defects and disrupts WNT/PCP genes and proteins in the developing heart. The successful candidate will combine
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using proteomics. (3) determine differences between DsbA proteins of two model organisms, E. coli and M. smegmatis (4) Establishing termini restraining for inhibitor-protein crystallography in
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(AD), mouse models, cell culture or imaging is highly desirable. The postdoctoral fellow will join a multidisciplinary and dynamic research team focused on understanding the role of microglia in
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and Professional Development | IU School of Medicine Basic Qualifications A recent PhD in immunology, virology, or a related field with a solid publication track record including at least 1 first author
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differences between DsbA proteins of two model organisms, E. coli and M. smegmatis (4) Establishing termini restraining for inhibitor-protein crystallography in collaboration with Prof. Weikai Li. Other general
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the genetic and developmental basis of pediatric heart disease. We analyze genetic variation in samples from patients with congenital heart defects and utilize mouse, Xenopus and cell-based models to assess
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been ranked among the top in the nation. The Department currently has an ongoing PhD program in Biostatistics, MS program in Biostatistics, and BS program in Health Data Science. We have established
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pathophysiology of the inner ear using multiple genetic mouse models and a novel human stem cell-derived 3D inner ear organoid model. We study genetically-mediated hair cell and neuronal degeneration in these model
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spatial transcriptomics to link molecular signatures with tissue architecture. Develop predictive models for disease diagnosis, prognosis, and therapeutic targeting. Experimental and Analytical Approaches