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of the lab is to characterize the role of nuclear pore complex machinery in mediating HIV-1 nuclear entry and subsequent downstream process that leads to productive viral gene integration. The goal
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— investigating how mechanical forces from focused ultrasound alter immune cell function and tumor microenvironments Acoustic tool development — designing next-generation transducers, wearable ultrasound devices
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multidisciplinary team of students and Ph.D.-level staff tasked with obtaining a molecular understanding bacterial sulfur metabolism and mechanisms of hydrogen sulfide (H2S)/reactive sulfur species (RSS) homeostasis
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characterize the role of nuclear pore complex machinery in mediating HIV-1 nuclear entry and subsequent downstream process that leads to productive viral gene integration. The goal of this position is to work
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studies the mechanisms by which chronic viral infections suppress antiviral immunity and drive T cell exhaustion to facilitate viral persistence. We also have a major interest in how chronic viral
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experimental disease models to identify disease mechanisms, discover therapeutic targets, and improve disease prediction, prevention, and treatment. Our research focuses primarily on substance use disorders and
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disease to elucidate these mechanisms. Duties • Administer and oversee the research projects. • Direct the preparation and execution of experiments to meet the needs of the project. • Assist in identifying
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, these efforts aim to transform human genetic diversity into a powerful resource for understanding complex disease mechanisms and advancing precision medicine. Responsibilities The successful candidate will have
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fellow to join the 3D Stem Cell Biology Research Lab (https://www.hashinolab.com ) and study normal and pathological development of the human inner ear using stem cell-derived organoids as a model system
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seeking a highly motivated postdoctoral fellow to investigate metabolic mechanisms of immune suppression in the prostate tumor microenvironment. Our research focuses on understanding how cancer cells