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, interlocking systems. We believe that access to nutritious food and a healthy environment is a fundamental human right. We believe that ensuring a prosperous global future depends on the ability to support local
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The University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | about 2 months ago
-based gene editing in primary human large- and small-airway epithelial cells. The Postdoctoral Research Associate may also contribute to studies of bronchiectasis, cystic fibrosis, asthma, and COPD, using
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of human disease, particularly neurodevelopmental disorders, including data analysis, image processing, literature research, and composition. They will handle transgenic mouse models, maintain colonies
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analyses Existing Sworn Security Status security clearance Salary and Benefits: Salary Range: $63,480 -$65,000. This position is eligible for Contract collegebenefits . To apply: https
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follow a bedside-to-bench template starting from gene variants already identified in human native and allograft kidney disease, to elucidating mechanisms via invitro tools to the development and/or
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of Program & Employment Compliance at [email protected] . For general application assistance or if you have questions about a job posting, please contact Human Resources at 501-671-2219 or 479-502
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position of which they have applied. For general application assistance or if you have questions about a job posting, please contact Human Resources at 479.575.5351. Department: Post Docs, GAs and Hourly
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”). Eligible new hires with prior URS service, may elect to enroll in URS if they make the election before they become eligible for retirement (usually the first day of work). Contact Human Resources at (801
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, which remains a major global health challenge and an important cause of liver related morbidity and mortality. Our laboratory has a strong history of developing innovative human models of viral infection
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Albert Einstein College of Medicine (Bronx, NY) | New York City, New York | United States | 5 days ago
, translational, and clinical studies to uncover mechanisms linking inflammation, DNA damage, and stem cell aging to cancer initiation and progression. Using murine and humanized models, human patient samples