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represents a central limitation to current anti-cancer immunotherapies and strategies aiming at reversing this process are still lacking. By exploring the intimate mechanisms regulating CD8 T cell exhaustion
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this process are still lacking. By exploring the intimate mechanisms regulating CD8 T cell exhaustion, our group aims to identify innovative opportunities to reverse this process and unleash the full potential
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bacteria and surface interaction at single-cell level and identification of the mode of mechanisms used by bacteria for mechanosensing and the effect of the material properties on bacterial viability
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fascinated by the intricate mechanisms governing cell fate transitions within the nucleus. Our mission is to decode how epigenetic regulation crafts neural development, leveraging brain organoids as our
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Sciences (DPhW), is offering a postdoc position to investigate mechanisms determining the sensitivity of immune cells towards endogenous and pharmacological glucocorticoids. The project is financed for two
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between nutrition, metabolism and the epigenome. The group uses a translational approach from in vitro systems and mice to humans combining next-generation sequencing, single-cell methodologies, genetics
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platform to investigate bacteria and surface interaction at single-cell level and identification of the mode of mechanisms used by bacteria for mechanosensing and the effect of the material properties