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Field
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-of-the-art facilities, explore fundamental plasma dynamics and non-equilibrium plasma chemistry inherent to air-breathing thrusters, and assess propulsion performance using industry-standard thrust stands
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. The ultimate goal of the lab is to understand the biology of these diseases, to define novel biomarkers, and to identify novel therapeutical targets. We have pioneered in the integration of genetics with omic
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System includes the University of North Texas in Denton and Frisco, the University of North Texas at Dallas and UNT Dallas College of Law, and the University of North Texas Health Science Center at Fort
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contribute to a team environment. Technical Proficiency: Skilled in using office software, technology, and relevant computer applications. Communication: Strong and clear written and verbal communication
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University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | about 2 months ago
); fibrin(ogen) degradation and the cellular and enzymatic drivers of thrombus resolution beyond plasminogen; and cellular and plasma mediators of FXI biology that influence bleeding risk. Additional
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models and machine learning techniques for kinetic equations arising from plasma and neutron transport. The position will be based at Virginia Tech’s campus in Blacksburg, VA. The postdoc will have a
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endoplasmic reticulum (ER) membrane. The project will combine cutting-edge approaches in functional genomics, mechanistic cell biology, cryo-electron microscopy, and protein engineering. We are particularly
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for all postdoctoral scholars appointed through the Office of Postdoctoral Affairs. The FY27 minimum is $79, 056. Our lab recently developed a new methylation profiling technology, FLEXseq (PMID: 42130071
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are not limited to the following projects: Genetics of Cardiometabolic Diseases in the Veteran Affairs Population(link is external) (I01 BX003362) Using genetic variation to study biology of blood lipids
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candidate will drive one of two major projects in the laboratory: 1) understanding how altered plasma membrane excitability contributes to muscle atrophy and the loss of motor units in aging and