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diseases and infections with antimicrobial resistance (AMR). Bacterial diarrheal pathogens contributes significantly to infant mortality, with many classified as WHO AMR priority pathogens. Interventions
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pathways and the antimicrobial resistance of causative pathogens. The study involves universities and public health agencies across the UK and uses advanced tools to detect infections and track resistance
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pathogens, which cause significant losses in crop yield each year. While controlling microbial pathogens is essential for sustainable agriculture and global food security, developing durable disease
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sensitivity, on resistance evolution. As model pathogen, we will focus on UTI E. coli and/or lung-infecting Pseudomonas aeruginosa. As a general reference, check out the group’s publications on AMR evolution
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Research Center Borstel - Leibniz Lung Center | Hamburg Gro Borstel, Hamburg | Germany | 2 months ago
mechanisms of bacterial plasticity in clinically relevant pathogens, including ESKAPE pathogens, mycobacteria, and other medically important species. As a PhD candidate, you will join the Leibniz ScienceCampus
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antigen-independent and can facilitate cross-pathogen protection. This project focuses on understanding how cells integrate distinct inflammatory cues and combinations thereof and how this leads
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hepatitis C virus, dengue virus, Zika virus, and norovirus — pathogens responsible for conditions such as liver cirrhosis, hepatocellular carcinoma, hemorrhagic fever, congenital birth defects, and severe
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join a young and dynamic research team investigating the molecular mechanisms of innate immunity, cell death signalling, and host-pathogen interactions. For more information about the lab, please visit
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with extracellular vesicles or host–pathogen systems (e.g. amoebae–bacteria interactions, including Legionella); and familiarity with metal/metalloid assays (e.g. As, Cu, Fe, Sb, Zn), disinfection by
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the role of the mitogen-activated protein kinase, Mps1, which plays an essential role in appressorium development and host penetration. Deletion of the MPS1 gene abolishes pathogenicity of the fungus. Very