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pathways are activated in Parkinson’s and contribute to neuronal dysfunction. The project will involve differentiating human iPS cells to microglia, astrocytes and dopaminergic neurons, establishing co
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, astrocytes, oligodendrocytes, and microglia. Develop complex neuroimmune co-culture systems incorporating immune cells into human CNS models. Apply CRISPR/Cas-based genome engineering technologies
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dementia. Specifically, the position involves maintaining and advancing a robust protocol for 3D bioprinting of iPSC derived neurons, astrocytes, and microglia to study the pathophysiology of AD and FTD in a
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(AD), mouse models, cell culture or imaging is highly desirable. The postdoctoral fellow will join a multidisciplinary and dynamic research team focused on understanding the role of microglia in
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). Along with regular cell culture work, will perform differentiation protocols for generation of dopaminergic neurons, astrocytes and microglia. Will also perform lentiviral production and transduction
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direct experience establishing multicellular 3D organoid or co-culture systems and differentiating cells into relevant cell types (e.g., M1- and M2-macrophages/microglia), alongside a solid conceptual
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proteomics, single-cell genomics, CRISPR genome engineering, and human iPSC-derived microglia and motor neuron models, our goal is to identify novel therapeutic targets that can be translated into effective
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neurodegeneration, microglial activation, microglia-vascular interactions and angiogenesis, tissue injury and repair, and therapeutic response. The position will integrate in vivo ocular disease models, primary
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brain tumors. The Escoubas Lab investigates bidirectional communication between neurons and microglia through nucleic acid sensing pathways, with implications for neurodevelopment, neurological disease
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mouse models relevant to MS pathogenesis and recovery. Performing and overseeing cellular and molecular studies involving macrophages, microglia, and other CNS-resident or infiltrating immune cells