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DNA methylation patterns, (iii) constructing, validating and characterizing polygenic risk profiles for PTSD, and (iv) identifying mechanisms underlying metabolic dysfunction in PTSD with a network
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dysregulated subnetworks that are related to glucocorticoid receptor regulation and Fc epsilon R1 signaling in PTSD based on changes in DNA methylation patterns, (iii) constructing, validating and characterizing
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