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drugs. We are seeking a dynamic and motivated candidate with expertise in mammalian cell culture, small animal (rodent) imaging to support data acquisition and qualitative analysis of the imaging studies
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explores genetic causes of neurological disorders and aim to develop therapies for these conditions with a high complementary approach, ranging from stem cell modelling to functional genomic. Information
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-prone and apoptosis-resistant neurons, and integrate iPSC-derived microglia from defined genetic backgrounds, including APOE3/3 and APOE4/4 lines. The work will combine live imaging, single-cell RNA
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application of immunology to drug discovery, together with a strong understanding of disease pathology and cell biology underlying immune-mediated inflammatory disease. Strong experience in laboratory-based
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skills and critical thinking. Experience working with cell culture, tissues and/or advanced experimental models of disease. Experience in relevant immunological, molecular and cellular techniques such as
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. The group explores genetic causes of neurological disorders and aim to develop therapies for these conditions with a high complementary approach, ranging from stem cell modelling to functional genomic
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modelling, and machine learning approaches to analyse large-scale datasets, including bulk and single-cell sequencing, gene expression arrays, proteomics, and metabolomics. Working closely with senior
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Experience in knockout generation using CRISPR Experience in cellular and molecular immunology techniques, including flow cytometry, ex vivo Treg expansion, intracellular cytokine assays, and functional immune
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on understanding how cellular metabolic processes shape the immune response in health and disease. A particular focus is on B cell metabolism, and more specifically the germinal centre reaction, which is responsible
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on understanding how cellular metabolic processes shape the immune response in health and disease. A particular focus is on B cell metabolism, and more specifically the germinal centre reaction, which is responsible