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and their environment depends on decoding of extracellular cues into intracellular signaling cascades that are integrated through dynamic and context-specific networks driving the cellular responses
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record of scholarly productivity, and the ability to work independently, yet collaboratively, in a multidisciplinary environment with clinicians, computer scientists, and statisticians, are strongly
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toxicants leading to vascular disease, including tobacco, vaping, and air pollution. Through the use of the gene-environment interaction . Major focus will be on a funded project to identify the drivers
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underlying environmental toxicant-induced cardiovascular diseases and gene-environment interactions, with a specific emphasis on identifying modifiers of response to known toxicants leading to vascular
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educational backgrounds (low-income households and disadvantaged educational environments) to apply. Applications will be reviewed starting on February 1, 2024. Applicants who have questions about the position
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the next generation of computational scientists in the rheumatic diseases. Access to Stanford’s scientific community and resources provides an unparalleled environment to perform high-impact translational
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antimicrobial mechanisms in infected tissues. We seek to recruit individuals who are passionate about advancing mechanistic understanding of infection biology and to provide a supportive environment for fellows
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human behaviors and environments impact well-being. Mentorship The postdoc fellow will be a member of the Stanford Media and Personality Lab in the Department of Communication. The postdoc will be
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databases, which include claims data and electronic health record data. Our research group prioritizes a collaborative and inclusive team environment. The principal investigator is an experienced mentor who
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of Us, UK Biobank, and MVP), TOPMed, dbGap, and the Stanford clinical research database. We offer close mentorship in a small lab environment along with opportunities for local, national, and