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of the best-characterised mechanisms of cellular senescence. We hypothesise that telomere shortening triggers a DDRdependent senescence in DC patients' BM-derived HSPC leading to severe BMF and proinflammatory
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genetic engineering technologies (including gene editing with CRISPR-Cas9) will be positively evaluated. Overall, candidates with a strong background in stem cell biology, telomere biology and/or immunology